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- Pancreatic Cancer -


General Information:

Names:
Wikipedia entry:
Dr. Ray Shahelien entry: 

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Observations:

Garcinol is found within the rinds of the Garcinia indica fruit, otherwise known as mangosteen.

Lots of intesting info:
http://www.pancan.org/section_research/pdf/news_and_updates/PC%20News%20and%20Updates%20-%20November%202011.pdf

University of Iowa study tests ketogenic diet for lung, pancreatic cancers
http://fyi.uiowa.edu/11/08/ui-study-diet-cancers/
Researchers with UI Health Care have received a two-year, $340,023 grant from the National Cancer
Institute to investigate whether a ketogenic diet (high fat, low carbohydrate) can increase the
effectiveness of radiation and chemotherapy for lung and pancreatic cancer. The researchers hope that
the ketogenic diet will deprive cancer cells of glucose and force them to rely on their flawed
mitochondrial metabolism, causing oxidative stress and making the cancer cells more susceptible to
chemotherapy and radiation.

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Known sources:

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Natural sources:

Mangosteen

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References:

J Oncol. 2012;2012:709739. Epub 2012 May 23.
Synergistic effect of garcinol and curcumin on antiproliferative and apoptotic activity in pancreatic cancer cells.
Parasramka MA, Gupta SV.
Source

Department of Nutrition and Food Science, College of Liberal Arts and Sciences, Wayne State University, 3009 Science Hall, Detroit, MI 48201, USA.
Abstract

Pancreatic cancer (PaCa) is a major health concern due to its aggressiveness and early metastasis. Current treatments for PaCa are limited by development of resistance against therapy. As an alternative strategy, we assessed the combinatorial effect of dietary compounds, garcinol and curcumin, on human PaCa cells (BxPC-3 and Panc-1). A significant (P < 0.05) dose-dependent reduction in cell viability and increase in apoptosis were observed in both cell lines as compared to untreated controls. A combination index (CI) value < 1, for a two-way comparison of curcumin and garcinol, suggests synergism. The potency (Dm) of the combination of garcinol and curcumin was 2 to 10 fold that of the individual agents. This indicates that curcumin and garcinol in combination exhibit a high level of synergism, with enhanced bioactivity, thereby reducing the required effective dose required for each individually. This combinatorial strategy may hold promise in future development of therapies against PaCa.

PMID:22685460 [PubMed] http://www.ncbi.nlm.nih.gov/pubmed/22685460
Free full text article: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3366245/

Myricetin induces pancreatic cancer cell death via the induction of apoptosis and inhibition of the phosphatidylinositol 3-kinase (PI3K) signaling pathway.
Cancer Lett. 2011 Sep 28;308(2):181-8. Epub 2011 Jun 14.
Phillips PA, Sangwan V, Borja-Cacho D, Dudeja V, Vickers SM, Saluja AK.
Source: Department of Surgery, University of Minnesota, Minneapolis, 55455, United States.
Abstract
Pancreatic cancer is the fourth leading cause of cancer related deaths and is a disease with poor prognosis. It is refractory to standard chemotherapeutic drugs or to novel treatment modalities, making it imperative to find new treatments. In this study, using both primary and metastatic pancreatic cancer cell lines, we have demonstrated that the flavonoid myricetin induced pancreatic cancer cell death in vitro via apoptosis, and caused a decrease in PI3 kinase activity. In vivo, treatment of orthotopic pancreatic tumors with myricetin resulted in tumor regression and decreased metastatic spread. Importantly, myricetin was non-toxic, both in vitro and in vivo, underscoring its use as a therapeutic agent against pancreatic cancer.
PMID: 21676539
http://www.ncbi.nlm.nih.gov/pubmed/21676539

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Updated: July 25, 2012
Inception: July 25, 2012